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failed to stimulated DuOx1/2 human promoter transcriptional activity in a rat thyroid cell line [ 3 ]. Despite its importance in thyroid hormone biosynthesis, no conclusive data about DuOx promoter regulation have been proposed. Thus, the current work
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Clin Endocrinol Metab 1999;84:476-486. 10.1210/jc.84.2.476 10022404 26 Clarke MJ, Erickson D, Castro MR, Atkinson JL: Thyroid-stimulating hormone pituitary adenomas. J Neurosurg 2008;109:17-22. 10.3171/JNS/2008/109/7/0017 18590428 27
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December 2009 in two iodine-sufficient areas of the Czech Republic – Prague ( n = 5520) and Vysočina region ( n = 2962). In total, 8482 women were examined for TSH (thyroid-stimulating hormone) and TPOAb (antibodies to thyroid peroxidase) in weeks 9–11 of
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Ruhr Center for Rare Diseases (CeSER), Ruhr University of Bochum and Witten/Herdecke University, Bochum, Germany
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thyroid gland, i.e. the stimulated amount of T 4 released per unit of time (termed ‘thyroid capacity', pmol/s), based on equilibrium levels of TSH and FT 4 as follows: Constants for hormone kinetics and plasma protein binding used in these
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TSH/IGF-1 Receptor Crosstalk in Graves’ Orbital Fibroblasts in Primary Culture The discovery of thyroid-stimulating hormone receptor (TSHR) expression in orbital fat tissue and extra-ocular muscles [ 1 ] suggested that TSHR is one of the
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) in the presence or absence of stimulation of thyroidal iodide uptake by treatment with a low-iodine diet (LID) for 1-4 weeks or exogenous thyroid-stimulating hormone (TSH) application prior to the administration of radioiodide [ 3 , 4 , 5 , 6 , 7 , 8
Department of Medicine, King Chulalongkorn Memorial Hospital, Bangkok, Thailand
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Department of Preventive and Social Medicine, Faculty of Medicine Siriraj Hospital, Mahidol University, Bangkok, Thailand
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Department of Preventive and Social Medicine, Faculty of Medicine Siriraj Hospital, Mahidol University, Bangkok, Thailand
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. Eligibility Criteria Our inclusion criteria were (1) published observational studies that addressed the association between NAFLD and thyroid hormone levels (free triiodothyronine [FT 3 ], free thyroxine [FT 4 ], or thyroid-stimulating hormone [TSH]) in
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Department of Endocrinology, Aalborg University Hospital, Aalborg, Denmark
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Department of Gynaecology and Obstetrics, Aalborg University Hospital, Aalborg, Denmark
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Department of Clinical Medicine, Aalborg University, Aalborg, Denmark
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Department of Clinical Medicine, Aalborg University, Aalborg, Denmark
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biochemical evaluation with the measurement of thyroid-stimulating hormone (TSH). Results revealed an elevated plasma level of TSH (>100 mIU/L). Simultaneous measurements of total thyroxine (T4) as well as total triiodothyronine (T3) showed that the plasma
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German Rheumatism Research Center, Berlin, Germany
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Institute for Clinical Chemistry, Interdisciplinary University Laboratory, University Medicine Göttingen, Göttingen, Germany
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[ 3 - 5 ]. Intra- and interindividual biological variations for laboratory blood markers including thyroid-stimulating hormone (TSH), free thyroxine (fT4), free triiodothyronine (fT3), and thyroglobulin are reported in Ricós’s table [ 6 ]. Observer and
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Introduction Thyroid hormones (TH) are critical in all phases of life and thus for development as well as for maintenance of homeostasis. The connection between thyroid dysfunction and disorders of the central nervous system (CNS), the