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  • iodine concentration level x
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Sylvie Hiéronimus Departments of Endocrinology, Diabetology and Reproductive Medicine

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Patricia Ferrari Departments of Biochemistry, University Hospital of Nice

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Jocelyn Gal Department of Biostatistics, University of Nice

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Frédéric Berthier Department of Biostatistics, University of Nice

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Stéphane Azoulay Institute of Chemistry, UMR 6001, University of Nice-Sophia-Antipolis

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André Bongain Gynaecology and Obstetrics, University Hospital of Nice

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Patrick Fénichel Departments of Endocrinology, Diabetology and Reproductive Medicine
Institut National de la Recherche Médicale, UMR U895, University Nice-Sophia-Antipolis, Nice, France

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Françoise Brucker-Davis Departments of Endocrinology, Diabetology and Reproductive Medicine
Institut National de la Recherche Médicale, UMR U895, University Nice-Sophia-Antipolis, Nice, France

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level [ 1 , 2 ], serum thyroglobulin (Tg) has been proposed as a marker of ID [ 3 ], including in newborns [ 4 , 5 ]. Maternal iodine supplementation has been shown to improve maternal and cord blood (CB) thyroid tests in areas of moderate and severe ID

Article Type:
Research Article
DOI:
https://doi.org/10.1159/000342915
Volume/Issue:
Volume 1: Issue 4
Free access
John H. Lazarus Thyroid Research Group, Institute of Molecular and Experimental Medicine, Cardiff University, University Hospital of Wales, Cardiff, UK

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supplements during gestation [ 14 ]. Interestingly, the frequency of elevated neonatal thyroid-stimulating hormone levels was still low (3%) in this population [ 15 ]. In Denmark, while the majority of pregnant women took iodine-containing supplements, the

Article Type:
Editorial
DOI:
https://doi.org/10.1159/000358873
Volume/Issue:
Volume 3: Issue 1
Free access

fortified by a universally set minimum amount of iodine. Adopting an agreed universal minimum concentration of salt iodine will allow the safe and effective improvement in the consumption of iodine throughout the EU. Individual member states would be allowed

Article Type:
Review Article
DOI:
https://doi.org/10.1159/000490143
Volume/Issue:
Volume 7: Issue 4
Free access
Bing Han Institute and Department of Endocrinology and Metabolism, Shanghai Ninth People’s Hospital Affiliated to Shanghai Jiaotong University School of Medicine, Shanghai, China

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Chi Chen Institute and Department of Endocrinology and Metabolism, Shanghai Ninth People’s Hospital Affiliated to Shanghai Jiaotong University School of Medicine, Shanghai, China

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Yi Chen Institute and Department of Endocrinology and Metabolism, Shanghai Ninth People’s Hospital Affiliated to Shanghai Jiaotong University School of Medicine, Shanghai, China

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Ningjian Wang Institute and Department of Endocrinology and Metabolism, Shanghai Ninth People’s Hospital Affiliated to Shanghai Jiaotong University School of Medicine, Shanghai, China

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Jie Yu Institute and Department of Endocrinology and Metabolism, Shanghai Ninth People’s Hospital Affiliated to Shanghai Jiaotong University School of Medicine, Shanghai, China

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Jing Cheng Institute and Department of Endocrinology and Metabolism, Shanghai Ninth People’s Hospital Affiliated to Shanghai Jiaotong University School of Medicine, Shanghai, China

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Yingchao Chen Institute and Department of Endocrinology and Metabolism, Shanghai Ninth People’s Hospital Affiliated to Shanghai Jiaotong University School of Medicine, Shanghai, China

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Chunfang Zhu Institute and Department of Endocrinology and Metabolism, Shanghai Ninth People’s Hospital Affiliated to Shanghai Jiaotong University School of Medicine, Shanghai, China

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Yingli Lu Institute and Department of Endocrinology and Metabolism, Shanghai Ninth People’s Hospital Affiliated to Shanghai Jiaotong University School of Medicine, Shanghai, China

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status has been determined to be more than adequate [ 7 ]. In another survey reported by the institute of iodine deficiency disorders, national median urine iodine concentration was 238.6, 184.4, and 174.4 μg/L in children, pregnant women, and lactating

Article Type:
Research Article
DOI:
https://doi.org/10.1159/000506701
Volume/Issue:
Volume 10: Issue 1
Free access
Françoise Brucker-Davis Department of Endocrinology, Diabetology and Reproductive Medicine
Institut National de la Recherche Médicale, UMR U895, Université Nice-Sophia Antipolis, Nice, France

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Patricia Panaïa-Ferrari Departments of Biochemistry, UMR U895, Université Nice-Sophia Antipolis, Nice, France

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Jocelyn Gal Departments of Biostatistics, CHU de Nice, UMR U895, Université Nice-Sophia Antipolis, Nice, France

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Patrick Fénichel Department of Endocrinology, Diabetology and Reproductive Medicine
Institut National de la Recherche Médicale, UMR U895, Université Nice-Sophia Antipolis, Nice, France

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Sylvie Hiéronimus Department of Endocrinology, Diabetology and Reproductive Medicine

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4) levels after the first trimester [ 1 ], which is considered by some as pathological, despite normal TSH concentration, and led to the concept of maternal hypothyroxinemia [ 2 ]. The definition of maternal hypothyroxinemia is usually set below the

Article Type:
Research Article
DOI:
https://doi.org/10.1159/000350882
Volume/Issue:
Volume 2: Issue 3
Free access
Katarzyna Pelewicz Department of Internal Medicine and Endocrinology, Medical University of Warsaw, Warsaw, Poland

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Rafał Wolny Department of Interventional Cardiology and Angiology, National Institute of Cardiology, Warsaw, Poland

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Tomasz Bednarczuk Department of Internal Medicine and Endocrinology, Medical University of Warsaw, Warsaw, Poland

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Piotr Miśkiewicz Department of Internal Medicine and Endocrinology, Medical University of Warsaw, Warsaw, Poland

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of free iodine and 15–60 g of bound iodine [ 2 ], an amount well above the acceptable upper level. In the case of excess iodine, thyroid discontinues the release of hormones (the acute Wolff-Chaikoff effect). This effect is usually transient, although

Article Type:
Research Article
DOI:
https://doi.org/10.1159/000515126
Volume/Issue:
Volume 10: Issue 4
Free access
Emilio Fiore Department of Experimental and Clinical Medicine, Endocrinology Unit 1, University Hospital of Pisa, Pisa, Italy

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Francesco Latrofa Department of Experimental and Clinical Medicine, Endocrinology Unit 1, University Hospital of Pisa, Pisa, Italy

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Paolo Vitti Department of Experimental and Clinical Medicine, Endocrinology Unit 1, University Hospital of Pisa, Pisa, Italy

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of iodine-induced thyrotoxicosis in subjects with preexisting autonomous multinodular goiter. This condition is transient and correlated to the level of iodine deficiency and the amount of iodine administered. In 1980, when in Switzerland the salt

Article Type:
Review Article
DOI:
https://doi.org/10.1159/000371741
Volume/Issue:
Volume 4: Issue 1
Free access
Christiaan F. Mooij Department of Pediatric Endocrinology, Emma Children’s Hospital, Amsterdam UMC, University of Amsterdam, Amsterdam, The Netherlands

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Nitash Zwaveling-Soonawala Department of Pediatric Endocrinology, Emma Children’s Hospital, Amsterdam UMC, University of Amsterdam, Amsterdam, The Netherlands

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Eric Fliers Department of Endocrinology and Metabolism, Amsterdam UMC, University of Amsterdam, Amsterdam, The Netherlands

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A.S. Paul van Trotsenburg Department of Pediatric Endocrinology, Emma Children’s Hospital, Amsterdam UMC, University of Amsterdam, Amsterdam, The Netherlands

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, while the other 2 patients reached normalization of FT3 only after 14 days of treatment. However, the use of amiodarone, an antiarrhythmic drug containing a high concentration of iodine, has not been described previously in the preoperative treatment

Article Type:
Letter
DOI:
https://doi.org/10.1159/000499104
Volume/Issue:
Volume 8: Issue 3
Open access
Paneeraq Noahsen Arctic Health Research Centre, Department of Clinical Medicine, Aalborg University, Aalborg, Denmark
Ilisimatusarfik, University of Greenland, Nuuk, Greenland
National Board of Health, Nuuk, Greenland

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Karsten F Rex Arctic Health Research Centre, Department of Clinical Medicine, Aalborg University, Aalborg, Denmark
Ilisimatusarfik, University of Greenland, Nuuk, Greenland
Department of Internal Medicine, Queen Ingrid’s Hospital, Nuuk, Greenland

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Inge Bülow Pedersen Department of Endocrinology, Aalborg University Hospital, Aalborg, Denmark

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Gert Mulvad Ilisimatusarfik, University of Greenland, Nuuk, Greenland
Queen Ingrid’s Health Care Centre, Nuuk, Greenland

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Hans Christian Florian-Sørensen Tasiilaq Health Care Center, Tasiilaq, Greenland

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Michael Lynge Pedersen Steno Diabetes Center Nuuk, Nuuk, Greenland

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Stig Andersen Arctic Health Research Centre, Department of Clinical Medicine, Aalborg University, Aalborg, Denmark
Ilisimatusarfik, University of Greenland, Nuuk, Greenland
Department of Internal Medicine, Queen Ingrid’s Hospital, Nuuk, Greenland
Department of Geriatric Medicine, Aalborg University Hospital, Aalborg, Denmark

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Introduction Iodine intake level is important for the occurrence of thyroid disorders ( 1 ). Low iodine intake is associated with an increased risk of goiter, thyroid dysfunction, and developmental brain damage if the iodine deficiency is

Article Type:
Research Article
Collections:
Thyroid autoimmunity
DOI:
https://doi.org/10.1530/ETJ-22-0071
Volume/Issue:
Volume 11: Issue 3
Open access
Rosália P. Padovani Thyroid Diseases Center and Laboratory of Molecular and Translational Endocrinology, Division of Endocrinology, Department of Medicine, Escola Paulista de Medicina, Universidade Federal de São Paulo, Brazil
Thyroid Diseases Center, Instituto Israelita de Ensino e Pesquisa Albert Einstein, Brazil
Department of Nuclear Medicine, Irmandade Santa Casa de Misericórdia de São Paulo, São Paulo, Brazil

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Rui M.B. Maciel Thyroid Diseases Center and Laboratory of Molecular and Translational Endocrinology, Division of Endocrinology, Department of Medicine, Escola Paulista de Medicina, Universidade Federal de São Paulo, Brazil
Thyroid Diseases Center, Instituto Israelita de Ensino e Pesquisa Albert Einstein, Brazil

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Teresa S. Kasamatsu Thyroid Diseases Center and Laboratory of Molecular and Translational Endocrinology, Division of Endocrinology, Department of Medicine, Escola Paulista de Medicina, Universidade Federal de São Paulo, Brazil

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Beatriz C.G. Freitas Thyroid Diseases Center and Laboratory of Molecular and Translational Endocrinology, Division of Endocrinology, Department of Medicine, Escola Paulista de Medicina, Universidade Federal de São Paulo, Brazil

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Marilia M.S. Marone Department of Nuclear Medicine, Irmandade Santa Casa de Misericórdia de São Paulo, São Paulo, Brazil

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Cleber P. Camacho Thyroid Diseases Center and Laboratory of Molecular and Translational Endocrinology, Division of Endocrinology, Department of Medicine, Escola Paulista de Medicina, Universidade Federal de São Paulo, Brazil
Thyroid Diseases Center, Instituto Israelita de Ensino e Pesquisa Albert Einstein, Brazil

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Rosa Paula M. Biscolla Thyroid Diseases Center and Laboratory of Molecular and Translational Endocrinology, Division of Endocrinology, Department of Medicine, Escola Paulista de Medicina, Universidade Federal de São Paulo, Brazil
Thyroid Diseases Center, Instituto Israelita de Ensino e Pesquisa Albert Einstein, Brazil

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challenging factor is the iodine uptake capacity of thyroid cancer tissue, which is poorer than that of normal thyroid cells [ 4 ]. In addition, to maximize the RAI uptake by residual thyroid remnants, persistent disease, or metastases, TSH levels must be

Article Type:
Research Article
DOI:
https://doi.org/10.1159/000433426
Volume/Issue:
Volume 4: Issue 2
Free access