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Institute of Public Health, University of Copenhagen, Odense, Denmark
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National Research Centre for the Working Environment, Copenhagen, Odense, Denmark
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Department of Palliative Medicine, Bispebjerg Hospital, Odense, Denmark
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symptoms of anxiety and depression disappeared within months of starting anti-thyroid drug treatment [ 2 , 14 , 15 ], indicating that increased levels of thyroid hormones may contribute to anxiety and depression [ 2 , 16 , 17 ]. Others found that symptoms
Department of Clinical Sciences, Skåne University Hospital, Malmö, Sweden
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Department of Clinical Sciences, Skåne University Hospital, Malmö, Sweden
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Department of Clinical Sciences, Skåne University Hospital, Malmö, Sweden
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other two treatment modalities, antithyroid drugs (ATD) or thyroidectomy, where TRAb slowly declines without a prior increase [ 10 ]. It has been suggested that the increase in TRAb after radioiodine treatment was mediated by a transient release of
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. Patients Case 1 This was a 52-year-old white Caucasian male smoker (15 pack-years) who had GD with unstable thyroid function for 2 years, treated with antithyroid drugs (ATD). In April 2009, he suddenly developed swelling of the right lid, right
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implications in the differential diagnosis of AITD, in predicting the outcome of GD after antithyroid drug treatment, in evaluating the risk of extrathyroidal manifestations of GD during pregnancy, and in predicting the likelihood of fetal/neonatal hyper- or
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.R.A.H.M.S AG, Berlin, Germany), and antithyroglobulin and antithyroid peroxidase antibodies. GD was treated with antithyroid drugs, either thiamazole or propylthiouracil, with no addition of L -tyrosine. The antithyroid drug was maintained according to the
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differ significantly from those obtained in the untreated samples. Sex hormone-binding globulin was elevated (228 nmol/l, normal range 30-60 nmol/l). TSH receptor antibodies were positive (14 IU/ml, normal range <2 IU/ml). Antithyroid peroxidase
Institute of Medicine, Huddinge, Karolinska Institutet, Stockholm, Sweden
Wallenberg’s Centre of Molecular and Translational Medicine, Region Västra Götaland, Sweden
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Institute of Medicine, Sahlgrenska Academy, University of Gothenburg, Göteborg, Sweden
Department of Endocrinology, Sahlgrenska University Hospital, Göteborg, Sweden
Gothenburg Centre for Person Centred-Care (GPCC), Göteborg, Sweden
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with the local treatment scheme for premenopausal female adult patients with GD where all patients received either antithyroid drugs (ATD) or surgery with previous ATD treatment. As an exception, one patient was treated with radioactive iodine
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multinodular goiter, and was well controlled by antithyroid drugs. The histology of the excised right ovarian mass revealed SO and the antithyroid treatment was therefore discontinued. In patients with SO causing thyrotoxicosis the hormonal profile does not
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antithyroid drug therapy. In case she developed mild GO, 1 in 4 respondents would add Se, and 50% of respondents would continue with Se also after remission of her Graves' hyperthyroidism. Based on a single study by Marcocci et al. [ 12 ], which showed
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Facultad de Ciencias Médicas, Instituto de Fisiología, Universidad Nacional de Cuyo, Mendoza, Argentina
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Facultad de Ciencias Exactas y Naturales, Universidad Nacional de Cuyo, Mendoza, Argentina
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surge of prostaglandins [ 11 ]. Lactation is another critical stage regulated by THs. Postnatal exposure to antithyroid drugs and hormones through the maternal feeding has important consequences for pups. Thus, we examined the indirect outcome of