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Kira Bang Bové Department of Endocrinology, Odense, Denmark

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Torquil Watt Department of Endocrinology, Odense, Denmark
Institute of Public Health, University of Copenhagen, Odense, Denmark

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Asmus Vogel Memory Disorders Research Group, Department of Neurology, Copenhagen University Hospital, Rigshospitalet, Odense, Denmark

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Laszlo Hegedüs Department of Endocrinology and Metabolism, Odense University Hospital, Odense, Denmark

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Jakob Bue Bjoerner Institute of Public Health, University of Copenhagen, Odense, Denmark
National Research Centre for the Working Environment, Copenhagen, Odense, Denmark

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Mogens Groenvold Institute of Public Health, University of Copenhagen, Odense, Denmark
Department of Palliative Medicine, Bispebjerg Hospital, Odense, Denmark

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Steen Joop Bonnema Department of Endocrinology and Metabolism, Odense University Hospital, Odense, Denmark

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Åse Krogh Rasmussen Department of Endocrinology, Odense, Denmark

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Ulla Feldt-Rasmussen Department of Endocrinology, Odense, Denmark

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symptoms of anxiety and depression disappeared within months of starting anti-thyroid drug treatment [ 2 , 14 , 15 ], indicating that increased levels of thyroid hormones may contribute to anxiety and depression [ 2 , 16 , 17 ]. Others found that symptoms

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Ola Lindgren Department of Endocrinology, Lund University, Malmö, Sweden
Department of Clinical Sciences, Skåne University Hospital, Malmö, Sweden

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Pernilla Asp Department of Oncology, Skåne University Hospital, Lund, Sweden

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Anna Sundlöv Department of Oncology, Lund University, and Department of Clinical Sciences, Skåne University Hospital, Lund, Sweden

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Jan Tennvall Department of Oncology, Lund University, and Department of Clinical Sciences, Skåne University Hospital, Lund, Sweden

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Bushra Shahida Department of Clinical Sciences Malmö, and Diabetes and Endocrinology, Lund University, Malmö, Sweden

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Tereza Planck Department of Endocrinology, Lund University, Malmö, Sweden
Department of Clinical Sciences, Skåne University Hospital, Malmö, Sweden

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Peter Åsman Department of Clinical Sciences Malmö, Ophthalmology, Lund University, and Department of Ophthalmology, Skåne University Hospital, Malmö, Sweden

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Mikael Lantz Department of Endocrinology, Lund University, Malmö, Sweden
Department of Clinical Sciences, Skåne University Hospital, Malmö, Sweden

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other two treatment modalities, antithyroid drugs (ATD) or thyroidectomy, where TRAb slowly declines without a prior increase [ 10 ]. It has been suggested that the increase in TRAb after radioiodine treatment was mediated by a transient release of

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C. Daumerie Departments of Endocrinology, Université catholique de Louvain, University Hospital St-Luc, Brussels, Belgium

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A. Boschi Departments of Ophthalmology, Université catholique de Louvain, University Hospital St-Luc, Brussels, Belgium

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P. Perros Department of Endocrinology, Royal Victoria Infirmary, Newcastle upon Tyne, UK

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. Patients Case 1 This was a 52-year-old white Caucasian male smoker (15 pack-years) who had GD with unstable thyroid function for 2 years, treated with antithyroid drugs (ATD). In April 2009, he suddenly developed swelling of the right lid, right

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Tanja Diana Molecular Thyroid Research Laboratory, Department of Medicine I, Johannes Gutenberg University (JGU) Medical Center, Mainz, Germany

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Christian Wüster Endocrine Laboratory Prof. Wüster, Mainz, Germany

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Paul D. Olivo Department of Microbiology, Washington University, St. Louis, Missouri, USA

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Angelica Unterrainer Molecular Thyroid Research Laboratory, Department of Medicine I, Johannes Gutenberg University (JGU) Medical Center, Mainz, Germany

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Jochem König Endocrine Laboratory Prof. Wüster, Mainz, Germany

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Michael Kanitz Molecular Thyroid Research Laboratory, Department of Medicine I, Johannes Gutenberg University (JGU) Medical Center, Mainz, Germany

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Artur Bossowski Department of Pediatrics, Endocrinology, and Diabetology, Medical University of Byalistok, Bialystok, Poland

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Brigitte Decallonne Division of Clinical and Experimental Endocrinology, UZ Leuven, Leuven, Belgium

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George J. Kahaly Molecular Thyroid Research Laboratory, Department of Medicine I, Johannes Gutenberg University (JGU) Medical Center, Mainz, Germany

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implications in the differential diagnosis of AITD, in predicting the outcome of GD after antithyroid drug treatment, in evaluating the risk of extrathyroidal manifestations of GD during pregnancy, and in predicting the likelihood of fetal/neonatal hyper- or

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Toshihide Yamamoto Yao Tokushukai General Hospital, Yao, Japan

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.R.A.H.M.S AG, Berlin, Germany), and antithyroglobulin and antithyroid peroxidase antibodies. GD was treated with antithyroid drugs, either thiamazole or propylthiouracil, with no addition of L -tyrosine. The antithyroid drug was maintained according to the

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Mahdi Kamoun Endocrinology and Metabolism Department

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Michèle d'Herbomez Department of Nuclear Medicine, Lille University Hospital, Lille

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Christine Lemaire Department of Endocrinology, Regional Hospital of Béthune, Béthune

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Armelle Fayard Endocrinology and Metabolism Department

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Rachel Desailloud Endocrinology and Metabolism Department, Amiens University Hospital, Amiens, France

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Damien Huglo Department of Nuclear Medicine, Lille University Hospital, Lille

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Jean-Louis Wemeau Endocrinology and Metabolism Department

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differ significantly from those obtained in the untreated samples. Sex hormone-binding globulin was elevated (228 nmol/l, normal range 30-60 nmol/l). TSH receptor antibodies were positive (14 IU/ml, normal range <2 IU/ml). Antithyroid peroxidase

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Mats Holmberg ANOVA, Karolinska University Hospital, Norra Stationsgatan 69, Stockholm, Sweden
Institute of Medicine, Huddinge, Karolinska Institutet, Stockholm, Sweden
Wallenberg’s Centre of Molecular and Translational Medicine, Region Västra Götaland, Sweden

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Helge Malmgren Institute of Medicine, Sahlgrenska Academy, University of Gothenburg, Göteborg, Sweden

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Peter F Berglund Institute of Neuroscience and Physiology, Department of Clinical Neuroscience, Sahlgrenska Academy, University of Gothenburg, Göteborg, Sweden

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Birgitta Johansson Institute of Medicine, Sahlgrenska Academy, University of Gothenburg, Göteborg, Sweden

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Helena Filipsson Nyström Wallenberg’s Centre of Molecular and Translational Medicine, Region Västra Götaland, Sweden
Institute of Medicine, Sahlgrenska Academy, University of Gothenburg, Göteborg, Sweden
Department of Endocrinology, Sahlgrenska University Hospital, Göteborg, Sweden
Gothenburg Centre for Person Centred-Care (GPCC), Göteborg, Sweden

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with the local treatment scheme for premenopausal female adult patients with GD where all patients received either antithyroid drugs (ATD) or surgery with previous ATD treatment. As an exception, one patient was treated with radioactive iodine

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Elli Anagnostou Departments of Endocrinology, Alexandra General Hospital, Athens, Greece

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Antonios Polymeris Departments of Endocrinology, Alexandra General Hospital, Athens, Greece

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Georgios Morphopoulos Departments of Pathology, Alexandra General Hospital, Athens, Greece

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Alexios Travlos Departments of Endocrinology, Alexandra General Hospital, Athens, Greece

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Vassiliki Sarantopoulou Departments of Endocrinology, Alexandra General Hospital, Athens, Greece

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Irini Papaspyrou Departments of Pathology, Alexandra General Hospital, Athens, Greece

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multinodular goiter, and was well controlled by antithyroid drugs. The histology of the excised right ovarian mass revealed SO and the antithyroid treatment was therefore discontinued. In patients with SO causing thyrotoxicosis the hormonal profile does not

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Laszlo Hegedüs Department of Endocrinology and Metabolism, Odense University Hospital, Odense, Denmark

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Steen J. Bonnema Department of Endocrinology and Metabolism, Odense University Hospital, Odense, Denmark

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Kristian H. Winther Department of Endocrinology and Metabolism, Odense University Hospital, Odense, Denmark

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antithyroid drug therapy. In case she developed mild GO, 1 in 4 respondents would add Se, and 50% of respondents would continue with Se also after remission of her Graves' hyperthyroidism. Based on a single study by Marcocci et al. [ 12 ], which showed

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Estefanía Rinaldini Consejo Nacional de Investigaciones Científicas y Técnicas (CONICET), Universidad Nacional de Cuyo, Mendoza, Argentina

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Fiorella Campo Verde Arboccó Consejo Nacional de Investigaciones Científicas y Técnicas (CONICET), Universidad Nacional de Cuyo, Mendoza, Argentina

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Marcelo Ezquer Centro de Medicina Regenerativa, Facultad de Medicina Clínica Alemana, Universidad del Desarrollo, Santiago, Chile

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Carlos Gamarra-Luques Consejo Nacional de Investigaciones Científicas y Técnicas (CONICET), Universidad Nacional de Cuyo, Mendoza, Argentina
Facultad de Ciencias Médicas, Instituto de Fisiología, Universidad Nacional de Cuyo, Mendoza, Argentina

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María Belén Hapon Consejo Nacional de Investigaciones Científicas y Técnicas (CONICET), Universidad Nacional de Cuyo, Mendoza, Argentina
Facultad de Ciencias Exactas y Naturales, Universidad Nacional de Cuyo, Mendoza, Argentina

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surge of prostaglandins [ 11 ]. Lactation is another critical stage regulated by THs. Postnatal exposure to antithyroid drugs and hormones through the maternal feeding has important consequences for pups. Thus, we examined the indirect outcome of

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