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into the follicular lumen, thereby both releasing TH from colloidal Tg and liberating entire Tg molecules from the matrix, prior to endocytosis ( 10 ). The balance between TH storage and release mediated by Tg is central for hormone yields and for the
Department of Endocrinology, Shengjing Hospital, China Medical University, Shenyang, Liaoning, China
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Department of Laboratory Medicine, The First Affiliated Hospital of China Medical University, Heping District, Shenyang, Liaoning, China
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Department of Endocrinology, Shengjing Hospital, China Medical University, Shenyang, Liaoning, China
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. TSH, TPOAb, and TgAb levels were measured via electrochemiluminescence immunoassays with a Cobas 601 analyzer (Roche Diagnostic). Free thyroxine (fT4) and free triiodothyronine (fT3) were measured when the participant’s TSH levels were outside the
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Department of Clinical Medicine, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark
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Department of Internal Medicine, Copenhagen University Hospital – Herlev and Gentofte, Copenhagen, Denmark
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Department of Public Health, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark
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were run through a diagnostic algorithm identifying all possible incident patients fulfilling the diagnostic criteria for biochemically overt hyper- or hypothyroidism, and a database of incident patients was established ( 31 ). Data were collected from