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Ilisimatusarfik, University of Greenland, Nuuk, Greenland
National Board of Health, Nuuk, Greenland
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Ilisimatusarfik, University of Greenland, Nuuk, Greenland
Department of Internal Medicine, Queen Ingrid’s Hospital, Nuuk, Greenland
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Queen Ingrid’s Health Care Centre, Nuuk, Greenland
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Ilisimatusarfik, University of Greenland, Nuuk, Greenland
Department of Internal Medicine, Queen Ingrid’s Hospital, Nuuk, Greenland
Department of Geriatric Medicine, Aalborg University Hospital, Aalborg, Denmark
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Introduction Iodine intake level is important for the occurrence of thyroid disorders ( 1 ). Low iodine intake is associated with an increased risk of goiter, thyroid dysfunction, and developmental brain damage if the iodine deficiency is
Institute of Molecular Pathology and Immunology of the University of Porto (IPATIMUP), Porto, Portugal
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Health Investigation and Innovation Institute (i3S), University of Porto, Porto, Portugal
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Health Investigation and Innovation Institute (i3S), University of Porto, Porto, Portugal
Public Health Unit, ACES Baixo Mondego, Coimbra, Portugal
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Department of Endocrinology, Hospital University Centre of São João, Porto, Portugal
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Faculty of Medicine of the University of Porto (FMUP), Porto, Portugal
Health Investigation and Innovation Institute (i3S), University of Porto, Porto, Portugal
Department of Pathology, Faculty of Medicine of the University of Porto, Porto, Portugal
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. ( 28 ) 2020 Observational 99 patients HT HT patients had lower Se levels than controls. Se-deficient patients exhibited higher TSH levels, thyroid volume, antibody titres, and urinary iodine compared to Se-sufficient ones ( P < 0
Department of Endocrinology and Metabolism, Institute of Endocrinology, NHC Key Laboratory of Diagnosis and Treatment of Thyroid Diseases, The First Affiliated Hospital of China Medical University, Shenyang, China
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of the subjects are shown in Table 1 , and time of thyroid tissue sample collection is provided in Supplementary Table 3. No significant differences in serum FT 3 , FT 4, and TSH concentrations were found between AIT and control group, while levels
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. Autoantibodies to TSHR are directly involved in the pathophysiology of AITD and their measurement is recommended for early diagnosis and management of patients with GD ( 9 ). Classical biochemical features of hyperthyroid GD, including elevated TH levels and