Asena Gökçay Canpolat
Asena Gökçay Canpolat
Department of Endocrinology and Metabolism, School of Medicine, Ankara University, Ankara, Turkey
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Murat Cinel
Murat Cinel
Department of Endocrinology and Metabolism, School of Medicine, Ankara University, Ankara, Turkey
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Serpil Dizbay Sak
Serpil Dizbay Sak
Department of Pathology, School of Medicine, Ankara University, Ankara, Turkey
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Işılay Taşkaldıran
Işılay Taşkaldıran
Department of Endocrinology and Metabolism, Ankara Training and Research Hospital, Ankara, Turkey
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Hakan Korkmaz
Hakan Korkmaz
Department of Endocrinology and Metabolism, School of Medicine, Süleyman Demirel University, Isparta, Turkey
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Özgür Demir
Özgür Demir
Department of Endocrinology and Metabolism, School of Medicine, Ankara University, Ankara, Turkey
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Reyhan Ersoy
Reyhan Ersoy
Department of Endocrinology and Metabolism, School of Medicine, Yıldırım Beyazıt University, Ankara, Turkey
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Selçuk Dağdelen
Selçuk Dağdelen
Department of Endocrinology and Metabolism, School of Medicine, Hacettepe University, Ankara, Turkey
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Dilek Berker
Dilek Berker
Department of Endocrinology and Metabolism, Ankara City Hospital, University of Health Sciences, Ankara, Turkey
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Klara Dalva
Klara Dalva
Department of Haematology, School of Medicine, Ankara University, Ankara, Turkey
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Adile Begüm Bahçecioğlu Mutlu
Adile Begüm Bahçecioğlu Mutlu
Department of Endocrinology and Metabolism, School of Medicine, Ankara University, Ankara, Turkey
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Murat Faik Erdoğan
Murat Faik Erdoğan
Department of Endocrinology and Metabolism, School of Medicine, Ankara University, Ankara, Turkey
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, desmin, p63, CD34, CD31, ICAM-1 antibody (Abcam®, Anti-ICAM-1 antibody [EPR4776]), and TGF-β antibody (Abcam®, Anti-TGF-β antibody [TGF-β17]) stains were performed. Flow Cytometric Study for Plasmablasts The ratio of plasmablast was detected in
Dulani Kottahachchi
Dulani Kottahachchi
Department of Endocrinology and Diabetes, The Alfred Hospital, Australia
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Duncan J. Topliss
Duncan J. Topliss
Department of Endocrinology and Diabetes, The Alfred Hospital, Australia
Department of Medicine, Monash University, Melbourne, VIC, Australia
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Department of Medicine, Monash University, Melbourne, VIC, Australia
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and their infiltration into tissue sites may be the cause of this disease state. Reactivation of these CD4+ CTL cells may require antigen presentation by plasmablasts or other activated B cells in tissue sites. Activated CD4+ CTLs presumably mediate
