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Lars Folkestad
Lars Folkestad
Department of Endocrinology, Odense University Hospital, Odense, Denmark
Institute of Clinical Research, University of Southern Denmark, Odense, Denmark
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Institute of Clinical Research, University of Southern Denmark, Odense, Denmark
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Frans Brandt
Frans Brandt
Department of Internal Medicine, Hospital of Southern Jutland, Sønderborg, Denmark
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Thomas Brix
Thomas Brix
Department of Endocrinology, Odense University Hospital, Odense, Denmark
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Marianne Vogsen
Marianne Vogsen
Department of Oncology, Odense University Hospital, Odense, Denmark
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Lars Bastholt
Lars Bastholt
Department of Oncology, Odense University Hospital, Odense, Denmark
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Peter Grupe
Peter Grupe
Department of Nuclear Medicine, Odense University Hospital, Odense, Denmark
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Jeanette Krogh Petersen
Jeanette Krogh Petersen
Department of Clinical Pathology, Odense University Hospital, Odense, Denmark
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Laszlo Hegedüs
Laszlo Hegedüs
Department of Endocrinology, Odense University Hospital, Odense, Denmark
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What Is Known about This Topic? The prevalence of thyrotoxicosis in patients with thyroid cancer is unknown. In view of the sparse reports, it is most likely very low. Thyrotropin receptor antibodies are seldom measured or found in
Tanja Diana
Tanja Diana
Molecular Thyroid Research Laboratory, Department of Medicine I, Johannes Gutenberg University (JGU) Medical Center, Mainz, Germany
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Hans-Peter Holthoff
Hans-Peter Holthoff
AdvanceCor GmbH, Martinsried, Germany
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Julia Fassbender
Julia Fassbender
AdvanceCor GmbH, Martinsried, Germany
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Christian Wüster
Christian Wüster
Endocrine Laboratory and Practice Prof. Wüster, Mainz, Germany
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Michael Kanitz
Michael Kanitz
Molecular Thyroid Research Laboratory, Department of Medicine I, Johannes Gutenberg University (JGU) Medical Center, Mainz, Germany
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George J. Kahaly
George J. Kahaly
Molecular Thyroid Research Laboratory, Department of Medicine I, Johannes Gutenberg University (JGU) Medical Center, Mainz, Germany
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Martin Ungerer
Martin Ungerer
AdvanceCor GmbH, Martinsried, Germany
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mice were also studied. Free Thyroxine Levels and Thyrotropin Receptor Antibodies Serum fT4 and TSHR-binding inhibiting autoantibodies (TBII) levels were measured with Immulite 2000 XPi (Siemens, Erlangen, Germany) according to the
Ringo Manta
Ringo Manta
Department of Nuclear Medicine, CHU Saint Pierre, Université Libre de Bruxelles (ULB), Brussels, Belgium
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Charlotte Martin
Charlotte Martin
Department of Infectious Diseases, CHU Saint Pierre, Université Libre de Bruxelles (ULB), Brussels, Belgium
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Vinciane Muls
Vinciane Muls
Department of Gastroenterology and Endoscopy, CHU Saint-Pierre, University Libre de Bruxelles (ULB), Brussels, Belgium
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Kris G Poppe
Kris G Poppe
Department of Endocrinology, CHU Saint Pierre, Université Libre de Bruxelles (ULB), Brussels, Belgium
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vedolizumab, oral budesonide and mycophenolic acid. In April 2020, the patient presented a mild COVID-19 and received hydroxychloroquine. Concomitantly, he developed transient hypothyroidism with negative thyroid peroxidase antibody (TPOAb) and thyrotropin-receptor
Meihua Jin
Meihua Jin
Division of Endocrinology and Metabolism, Department of Internal Medicine, Asan Medical Center, University of Ulsan College of Medicine, Seoul, Republic of Korea
Division of Endocrinology and Metabolism, Department of Internal Medicine, Korea University College of Medicine and School of Medicine, Seoul, Republic of Korea
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Division of Endocrinology and Metabolism, Department of Internal Medicine, Korea University College of Medicine and School of Medicine, Seoul, Republic of Korea
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Ahreum Jang
Ahreum Jang
Division of Endocrinology and Metabolism, Department of Internal Medicine, Asan Medical Center, University of Ulsan College of Medicine, Seoul, Republic of Korea
Division of Endocrinology and Metabolism, Department Internal Medicine, Dankook University College of Medicine, Cheonan, Republic of Korea
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Division of Endocrinology and Metabolism, Department Internal Medicine, Dankook University College of Medicine, Cheonan, Republic of Korea
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Chae A Kim
Chae A Kim
Division of Endocrinology and Metabolism, Department of Internal Medicine, Asan Medical Center, University of Ulsan College of Medicine, Seoul, Republic of Korea
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Tae Yong Kim
Tae Yong Kim
Division of Endocrinology and Metabolism, Department of Internal Medicine, Asan Medical Center, University of Ulsan College of Medicine, Seoul, Republic of Korea
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Won Bae Kim
Won Bae Kim
Division of Endocrinology and Metabolism, Department of Internal Medicine, Asan Medical Center, University of Ulsan College of Medicine, Seoul, Republic of Korea
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Young Kee Shong
Young Kee Shong
Division of Endocrinology and Metabolism, Department of Internal Medicine, Asan Medical Center, University of Ulsan College of Medicine, Seoul, Republic of Korea
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Min Ji Jeon
Min Ji Jeon
Division of Endocrinology and Metabolism, Department of Internal Medicine, Asan Medical Center, University of Ulsan College of Medicine, Seoul, Republic of Korea
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Won Gu Kim
Won Gu Kim
Division of Endocrinology and Metabolism, Department of Internal Medicine, Asan Medical Center, University of Ulsan College of Medicine, Seoul, Republic of Korea
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-Tc thyroid scan and uptake are performed in 728 patients. ATD, antithyroid drug; CAM, carbimazole; MMDT, minimum maintenance dose therapy; MMI, methimazole; PTU, propylthiouracil; TAO, thyroid-associated ophthalmopathy; TRAb, thyrotropin receptor antibody
John H. Lazarus
John H. Lazarus
Centre for Endocrine and Diabetes Sciences, Cardiff University, Cardiff, UK
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;83:514–518. 10.1210/jc.83.2.514 9467567 5 Amino N, Izumi Y, Hidaka Y, Takeoka K, Nakata Y, Tatsumi KI, Nagata A, Takano T: No increase of blocking type anti-thyrotropin receptor antibodies during pregnancy in patients with Graves’ disease. J Clin
Tanja Diana
Tanja Diana
Molecular Thyroid Research Laboratory, Department of Medicine I, Johannes Gutenberg University (JGU) Medical Center, Mainz, Germany
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Paul D. Olivo
Paul D. Olivo
Department of Molecular Microbiology, Washington University Medical School, St. Louis, Missouri, USA
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Yie-Hwa Chang
Yie-Hwa Chang
Mediomics, LLC, St. Louis, Missouri, USA
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Christian Wüster
Christian Wüster
Endocrine Laboratory and Practice Prof. Wüster, Mainz, Germany
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Michael Kanitz
Michael Kanitz
Molecular Thyroid Research Laboratory, Department of Medicine I, Johannes Gutenberg University (JGU) Medical Center, Mainz, Germany
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George J. Kahaly
George J. Kahaly
Molecular Thyroid Research Laboratory, Department of Medicine I, Johannes Gutenberg University (JGU) Medical Center, Mainz, Germany
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-9303 11 Kahaly GJ , Wüster C , Olivo PD , Diana T . High Titers of Thyrotropin Receptor Antibodies Are Associated With Orbitopathy in Patients With Graves Disease . J Clin Endocrinol Metab . 2019 Jul ; 104 ( 7 ): 2561 – 8 . 10.1210/jc
Tereza Planck
Tereza Planck
Department of Clinical Sciences Malmö, Lund University, Malmö, Sweden
Department of Endocrinology, Skåne University Hospital, Malmö, Sweden
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Department of Endocrinology, Skåne University Hospital, Malmö, Sweden
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Bushra Shahida
Bushra Shahida
Department of Clinical Sciences Malmö, Lund University, Malmö, Sweden
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Johan Malm
Johan Malm
Department of Translational Medicine, Section for Clinical Chemistry, Lund University, Malmö, Sweden
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Jonas Manjer
Jonas Manjer
Department of Clinical Sciences Malmö, Lund University, Malmö, Sweden
Department of Surgery, Skåne University Hospital, Malmö, Sweden
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Department of Surgery, Skåne University Hospital, Malmö, Sweden
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antibodies (TPOAb), and thyrotropin receptor antibodies (TRAb); (ii) the presence of GO; and (iii) relapse within 1 year after terminating treatment with antithyroid drugs. Regarding the analysis of relapse, 139 patients were treated with antithyroid drugs as
Earn H. Gan
Earn H. Gan
Institute of Genetic Medicine, International Centre for Life, Newcastle upon Tyne, United Kingdom
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Anna L. Mitchell
Anna L. Mitchell
Institute of Genetic Medicine, International Centre for Life, Newcastle upon Tyne, United Kingdom
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Ruth Plummer
Ruth Plummer
Institute of Genetic Medicine, International Centre for Life, Newcastle upon Tyne, United Kingdom
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Simon Pearce
Simon Pearce
Institute of Genetic Medicine, International Centre for Life, Newcastle upon Tyne, United Kingdom
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Petros Perros
Petros Perros
Institute of Genetic Medicine, International Centre for Life, Newcastle upon Tyne, United Kingdom
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.5–21.5). Thyroid peroxidase antibodies were elevated at >600 kU/L (reference range 0–34), as was the thyrotropin receptor antibody level, measured at 5.0 IU/L (reference range1.0–1.8; BRAHMS Kryptor TRAK assay), consistent with Graves disease (Fig. 1 ). The
Emna Jelloul
Emna Jelloul
Endocrine Unit Centre Hospitalier Universitaire (CHU) Saint Pierre, Université Libre de Bruxelles (ULB), Rue Haute, Brussels, Belgium
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Georgiana Sitoris
Georgiana Sitoris
Endocrine Unit Centre Hospitalier Universitaire (CHU) Saint Pierre, Université Libre de Bruxelles (ULB), Rue Haute, Brussels, Belgium
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Flora Veltri
Flora Veltri
Endocrine Unit Centre Hospitalier Universitaire (CHU) Saint Pierre, Université Libre de Bruxelles (ULB), Rue Haute, Brussels, Belgium
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Pierre Kleynen
Pierre Kleynen
Endocrine Unit Centre Hospitalier Universitaire (CHU) Saint Pierre, Université Libre de Bruxelles (ULB), Rue Haute, Brussels, Belgium
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Serge Rozenberg
Serge Rozenberg
Departement of Gynecology and Obstetrics, Centre Hospitalier Universitaire Saint Pierre, Université Libre de Bruxelles (ULB), Rue Haute, Brussels, Belgium
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Kris G Poppe
Kris G Poppe
Endocrine Unit Centre Hospitalier Universitaire (CHU) Saint Pierre, Université Libre de Bruxelles (ULB), Rue Haute, Brussels, Belgium
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TSH measurement was available (initially at 11.3 ± 2.15 weeks versus 18.9 ± 4.3 for the control) and from them, 29 (87.9%) normalised their TSH levels. In 19 of the 36 women (52.8%), thyrotropin receptor antibodies (TSH-R-Ab) were measured and negative
Marta Nascimento Soares
Marta Nascimento Soares
Faculty of Medicine of the University of Porto, Porto, Portugal
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Marta Borges-Canha
Marta Borges-Canha
Faculty of Medicine of the University of Porto, Porto, Portugal
Department of Endocrinology, Diabetes and Metabolism, Centro Hospitalar Universitário de São João, Porto, Portugal
Department of Surgery and Physiology, Cardiovascular Research Unit, Faculty of Medicine from the University of Porto, Porto, Portugal
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Department of Endocrinology, Diabetes and Metabolism, Centro Hospitalar Universitário de São João, Porto, Portugal
Department of Surgery and Physiology, Cardiovascular Research Unit, Faculty of Medicine from the University of Porto, Porto, Portugal
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Celestino Neves
Celestino Neves
Faculty of Medicine of the University of Porto, Porto, Portugal
Department of Endocrinology, Diabetes and Metabolism, Centro Hospitalar Universitário de São João, Porto, Portugal
Institute for Research Innovation in Health, University of Porto, Porto, Portugal
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Department of Endocrinology, Diabetes and Metabolism, Centro Hospitalar Universitário de São João, Porto, Portugal
Institute for Research Innovation in Health, University of Porto, Porto, Portugal
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João Sérgio Neves
João Sérgio Neves
Faculty of Medicine of the University of Porto, Porto, Portugal
Department of Endocrinology, Diabetes and Metabolism, Centro Hospitalar Universitário de São João, Porto, Portugal
Department of Surgery and Physiology, Cardiovascular Research Unit, Faculty of Medicine from the University of Porto, Porto, Portugal
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Department of Endocrinology, Diabetes and Metabolism, Centro Hospitalar Universitário de São João, Porto, Portugal
Department of Surgery and Physiology, Cardiovascular Research Unit, Faculty of Medicine from the University of Porto, Porto, Portugal
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Davide Carvalho
Davide Carvalho
Faculty of Medicine of the University of Porto, Porto, Portugal
Department of Endocrinology, Diabetes and Metabolism, Centro Hospitalar Universitário de São João, Porto, Portugal
Institute for Research Innovation in Health, University of Porto, Porto, Portugal
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Department of Endocrinology, Diabetes and Metabolism, Centro Hospitalar Universitário de São João, Porto, Portugal
Institute for Research Innovation in Health, University of Porto, Porto, Portugal
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Introduction Graves' disease is an autoimmune thyroid disease, being the most frequent cause of hyperthyroidism ( 1 ). It is caused by the production of autoantibodies (thyrotropin receptor antibodies (TRAbs)) against the thyrotropin receptor
