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Carla Colombo Endocrine Oncology Unit, Department of Endocrine and Metabolic Diseases, Istituto Auxologico Italiano IRCCS, Milan, Italy
Department of Pathophysiology and Transplantation, University of Milan, Milan, Italy

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Daniele Ceruti Department of Biotechnology and Translational Medicine, University of Milan, Milan, Italy

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Massimiliano Succi Department of Biotechnology and Translational Medicine, University of Milan, Milan, Italy

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Simone De Leo Endocrine Oncology Unit, Department of Endocrine and Metabolic Diseases, Istituto Auxologico Italiano IRCCS, Milan, Italy

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Matteo Trevisan Endocrine Oncology Unit, Department of Endocrine and Metabolic Diseases, Istituto Auxologico Italiano IRCCS, Milan, Italy
Department of Biotechnology and Translational Medicine, University of Milan, Milan, Italy

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Claudia Moneta Department of Biotechnology and Translational Medicine, University of Milan, Milan, Italy

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Laura Fugazzola Endocrine Oncology Unit, Department of Endocrine and Metabolic Diseases, Istituto Auxologico Italiano IRCCS, Milan, Italy
Department of Pathophysiology and Transplantation, University of Milan, Milan, Italy

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, respectively, and often leads to treatment discontinuation or withdrawal with a possible reduction in efficacy and in progression-free survival (PFS) ( 14 , 15 ). In 2018, for the first time, we demonstrated a correlation between fatigue and primary adrenal

Article Type:
Research Article
Collections:
Thyroid cancer - basic and translational
DOI:
https://doi.org/10.1530/ETJ-23-0246
Volume/Issue:
Volume 13: Issue 3
Open access
Toshihide Yamamoto Yao Tokushukai General Hospital, Yao, Japan

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Introduction Variable occurrence of autoimmune thyroid disease (ATD) in patients with primary adrenal insufficiency (PAI) has been reported: 24% of 148 patients with Addison's disease in Poland [ 1 ], 47% of 664 patients in Norway [ 2 ], and

Article Type:
Case Report
DOI:
https://doi.org/10.1159/000433532
Volume/Issue:
Volume 4: Issue 3
Free access
Sofie Jespersen Department of Internal Medicine and Endocrinology, Copenhagen University Hospital, Herlev, Denmark

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Birte Nygaard Department of Internal Medicine and Endocrinology, Copenhagen University Hospital, Herlev, Denmark

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Lars Østergaard Kristensen Department of Internal Medicine and Endocrinology, Copenhagen University Hospital, Herlev, Denmark

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glucocorticoid treatment is the suppression of the hypothalamic-pituitary-adrenal (HPA) axis with impairment of endogenous cortisol production, implicating symptoms of adrenocortical insufficiency, especially in the period after cessation of therapy with possible

Article Type:
Research Article
DOI:
https://doi.org/10.1159/000440834
Volume/Issue:
Volume 4: Issue 4
Free access
Haggi Mazeh Department of Surgery, Jerusalem, Israel

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Amir Orlev Department of Surgery, Jerusalem, Israel

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Ido Mizrahi Department of Surgery, Jerusalem, Israel

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David J. Gross Endocrinology and Metabolism Service Hadassah-Hebrew University Medical Center, Jerusalem, Israel

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Herbert R. Freund Department of Surgery, Jerusalem, Israel

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, FSH, and testosterone and urinary free cortisol levels were all within normal limits. Urinary dopamine, epinephrine and norepinephrine levels were normal. Abdominal and pelvic CT scan revealed a lobular mass 2 × 2.4 cm superior to the left adrenal

Article Type:
Case Report
DOI:
https://doi.org/10.1159/000368750
Volume/Issue:
Volume 4: Issue 1
Free access
Hsu-Hua Tseng Department of Internal Medicine, National Taiwan University Hospital, Taipei, Taiwan

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Yen-Bo Lin Division of Endocrinology and Metabolism, Department of Internal Medicine, National Taiwan University Hospital, Bei-Hu Branch, Taipei, Taiwan

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Kuan-Yu Lin Division of Endocrinology and Metabolism, Department of Internal Medicine, National Taiwan University Hospital, Yun-Lin Branch, Douliu City, Taiwan

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Chia-Hung Lin Division of Endocrinology and Metabolism, Department of Internal Medicine, National Taiwan University Hospital, Hsin-Chu Branch, Hsin-Chu, Taiwan

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Hung-Yuan Li Division of Endocrinology and Metabolism, Department of Internal Medicine, National Taiwan University Hospital, Taipei, Taiwan

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Chia-Hsuin Chang Division of Endocrinology and Metabolism, Department of Internal Medicine, National Taiwan University Hospital, Taipei, Taiwan

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Yi-Ching Tung Department of Pediatrics, National Taiwan University Hospital, Taipei, Taiwan

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Pei-Lung Chen Division of Endocrinology and Metabolism, Department of Internal Medicine, National Taiwan University Hospital, Taipei, Taiwan
Graduate Institute of Medical Genomics and Proteomics, National Taiwan University College of Medicine, Taipei, Taiwan
Department of Medical Genetics, National Taiwan University Hospital, Taipei, Taiwan

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Chih-Yuan Wang Division of Endocrinology and Metabolism, Department of Internal Medicine, National Taiwan University Hospital, Taipei, Taiwan
Department of Internal Medicine, National Taiwan University College of Medicine, Taipei, Taiwan

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Wei-Shiung Yang Division of Endocrinology and Metabolism, Department of Internal Medicine, National Taiwan University Hospital, Taipei, Taiwan
Graduate Institute of Clinical Medicine, National Taiwan University College of Medicine, Taipei, Taiwan

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Shyang-Rong Shih Division of Endocrinology and Metabolism, Department of Internal Medicine, National Taiwan University Hospital, Taipei, Taiwan
Department of Internal Medicine, National Taiwan University College of Medicine, Taipei, Taiwan
Center of Anti-Aging and Health Consultation, National Taiwan University Hospital, Taipei, Taiwan

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Introduction Autoimmune polyendocrine syndrome (APS) was initially described in France by Claude and Gourgerot in 1908 ( 1 ). It is characterized by immunopathological involvement of at least two endocrine organs, such as the adrenal gland

Article Type:
Research Article
Collections:
Thyroid autoimmunity
DOI:
https://doi.org/10.1530/ETJ-23-0162
Volume/Issue:
Volume 12: Issue 6
Open access
Zoe A. Efstathiadou Department of Endocrinology, ‘Hippokration' General Hospital of Thessaloniki, Thessaloniki, Greece

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Albana Sykja Department of Endocrinology, ‘Hippokration' General Hospital of Thessaloniki, Thessaloniki, Greece

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Panagiotis Anagnostis Department of Endocrinology, ‘Hippokration' General Hospital of Thessaloniki, Thessaloniki, Greece

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Athanasios Panagiotou Department of Endocrinology, ‘Hippokration' General Hospital of Thessaloniki, Thessaloniki, Greece

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Marina Kita Department of Endocrinology, ‘Hippokration' General Hospital of Thessaloniki, Thessaloniki, Greece

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endogenous hypercortisolism also predisposes to thyrolytic processes not traditionally related to thyroid autoimmunity, especially while the hypothalamus-pituitary-adrenal axis is still suppressed. Introduction Exacerbation and increased prevalence of

Article Type:
Case Report
DOI:
https://doi.org/10.1159/000360396
Volume/Issue:
Volume 3: Issue 1
Free access
Minoru Kihara Departments of Surgery, Kobe, Japan

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Akira Miyauchi Departments of Surgery, Kobe, Japan

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Hiroshi Yoshida Research, Kuma Hospital, Kobe, Japan

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Osamu Yamada Departments of Surgery, Kobe, Japan

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Hiroo Masuoka Departments of Surgery, Kobe, Japan

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Tomonori Yabuta Departments of Surgery, Kobe, Japan

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Takuya Higashiyama Departments of Surgery, Kobe, Japan

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Mitsuhiro Fukushima Departments of Surgery, Kobe, Japan

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Yasuhiro Ito Departments of Surgery, Kobe, Japan

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Kaoru Kobayashi Departments of Surgery, Kobe, Japan

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Akihiro Miya Departments of Surgery, Kobe, Japan

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transmitted in an autosomal dominant pattern. MEN 2B is characterized by the presence of MTC, adrenal pheochromocytoma, mucosal neuromatosis, and a marfanoid habitus. About 95% of patients with MEN 2B carry a germline M918T mutation in the RET proto

Article Type:
Research Article
DOI:
https://doi.org/10.1159/000369134
Volume/Issue:
Volume 3: Issue 4
Free access
Francesca Orsolini Department of Clinical and Experimental Medicine, Endocrine Unit, University of Pisa, Pisa, Italy

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Alessandro Prete Department of Clinical and Experimental Medicine, Endocrine Unit, University of Pisa, Pisa, Italy

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Pierpaolo Falcetta Department of Clinical and Experimental Medicine, Endocrine Unit, University of Pisa, Pisa, Italy

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Domenico Canale Department of Clinical and Experimental Medicine, Endocrine Unit, University of Pisa, Pisa, Italy

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Fulvio Basolo Department of Surgical, Medical and Molecular Pathology and Critical Care Medicine, University of Pisa, Pisa, Italy

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Greta Alì Department of Surgical, Medical and Molecular Pathology and Critical Care Medicine, University of Pisa, Pisa, Italy

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Francesca Manassero Division of Urology, Department of Translational Research and of New Surgical and Medical Technologies, University of Pisa, Pisa, Italy

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Paolo Vitti Department of Clinical and Experimental Medicine, Endocrine Unit, University of Pisa, Pisa, Italy

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Rossella Elisei Department of Clinical and Experimental Medicine, Endocrine Unit, University of Pisa, Pisa, Italy

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Eleonora Molinaro Department of Clinical and Experimental Medicine, Endocrine Unit, University of Pisa, Pisa, Italy

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( 2 , 3 , 5 ). Other than to the soft tissues of the neck region, MTC spreads first to the liver (45%) and bone (45%) and less commonly to lung (33%), brain (1–5%) or skin (few cases reported) ( 6 ). Metastases to adrenal glands, pleura, heart, ovary

Article Type:
Case Report
Collections:
Thyroid cancer - clinical
DOI:
https://doi.org/10.1530/ETJ-21-0016
Volume/Issue:
Volume 11: Issue 2
Open access
Pedro Weslley Souza Rosario Santa Casa de Belo Horizonte, Belo Horizonte, Brazil

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Gabriela Franco Mourão Santa Casa de Belo Horizonte, Belo Horizonte, Brazil

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Maria Regina Calsolari Santa Casa de Belo Horizonte, Belo Horizonte, Brazil

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individuals with hyperthyrotropinemia unrelated to thyroid insufficiency [ 5 ]. Some conditions that can elevate serum TSH can be recognized easily (untreated adrenal insufficiency, chronic renal failure, antidopaminergic drugs, recovery from severe acute

Article Type:
Letter
DOI:
https://doi.org/10.1159/000441416
Volume/Issue:
Volume 4: Issue 4
Free access
Mikael Nilsson University of Gothenburg, Göteborg, Sweden

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E. Dillwyn Williams University of Cambridge, Cambridge, UK

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associated with sarcomas and cancers of the breast, brain and adrenal glands, and with leukaemias. Similarly, the RET gene is associated with lesions of both neural crest origin (neuroma and phaeochromocytoma) and of endodermal origin (medullary carcinoma

Article Type:
Letter
DOI:
https://doi.org/10.1159/000450680
Volume/Issue:
Volume 5: Issue 4
Free access