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  • Intercellular adhesion molecule-1 x
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Mahdi Kamoun Endocrinology and Metabolism Department

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Michèle d'Herbomez Department of Nuclear Medicine, Lille University Hospital, Lille

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Christine Lemaire Department of Endocrinology, Regional Hospital of Béthune, Béthune

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Armelle Fayard Endocrinology and Metabolism Department

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Rachel Desailloud Endocrinology and Metabolism Department, Amiens University Hospital, Amiens, France

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Damien Huglo Department of Nuclear Medicine, Lille University Hospital, Lille

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Jean-Louis Wemeau Endocrinology and Metabolism Department

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expression in thyrocytes. Fas antigen is functionally expressed on the surface of thyrocytes, and TSH inhibits Fas antigen-mediated apoptosis of thyrocytes resulting in the promotion of growth of the thyroid gland [ 15 ]. Intercellular adhesion molecule-1 and

Article Type:
Case Report
DOI:
https://doi.org/10.1159/000355386
Volume/Issue:
Volume 3: Issue 1
Free access
Sita Virakul Departments of Immunology, University Medical Center Rotterdam
Internal Medicine, Erasmus MC, University Medical Center Rotterdam

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Leendert van Steensel Departments of Immunology, University Medical Center Rotterdam

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Virgil A.S.H. Dalm Departments of Immunology, University Medical Center Rotterdam

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Dion Paridaens Rotterdam Eye Hospital, Rotterdam, The Netherlands
Department of Ophthalmology, Geneva University Hospitals, Geneva, Switzerland

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P. Martin van Hagen Departments of Immunology, University Medical Center Rotterdam
Internal Medicine, Erasmus MC, University Medical Center Rotterdam
Rotterdam Eye Hospital, Rotterdam, The Netherlands

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Willem A. Dik Departments of Immunology, University Medical Center Rotterdam

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1999;84:4079-4084. 10.1210/jcem.84.11.6111 10566653 33 Yan SX, Wang Y: Inhibitory effects of Triptolide on interferon-gamma-induced human leucocyte antigen-DR, intercellular adhesion molecule-1, CD40 expression on retro-ocular fibroblasts

Article Type:
Review Article
DOI:
https://doi.org/10.1159/000367968
Volume/Issue:
Volume 3: Issue 4
Free access
Asena Gökçay Canpolat Department of Endocrinology and Metabolism, School of Medicine, Ankara University, Ankara, Turkey

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Murat Cinel Department of Endocrinology and Metabolism, School of Medicine, Ankara University, Ankara, Turkey

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Serpil Dizbay Sak Department of Pathology, School of Medicine, Ankara University, Ankara, Turkey

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Işılay Taşkaldıran Department of Endocrinology and Metabolism, Ankara Training and Research Hospital, Ankara, Turkey

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Hakan Korkmaz Department of Endocrinology and Metabolism, School of Medicine, Süleyman Demirel University, Isparta, Turkey

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Özgür Demir Department of Endocrinology and Metabolism, School of Medicine, Ankara University, Ankara, Turkey

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Reyhan Ersoy Department of Endocrinology and Metabolism, School of Medicine, Yıldırım Beyazıt University, Ankara, Turkey

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Selçuk Dağdelen Department of Endocrinology and Metabolism, School of Medicine, Hacettepe University, Ankara, Turkey

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Dilek Berker Department of Endocrinology and Metabolism, Ankara City Hospital, University of Health Sciences, Ankara, Turkey

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Klara Dalva Department of Haematology, School of Medicine, Ankara University, Ankara, Turkey

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Adile Begüm Bahçecioğlu Mutlu Department of Endocrinology and Metabolism, School of Medicine, Ankara University, Ankara, Turkey

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Murat Faik Erdoğan Department of Endocrinology and Metabolism, School of Medicine, Ankara University, Ankara, Turkey

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in sera were analyzed. Serum fibrosis markers such as Intercellular Adhesion Molecule-1 (Boster® Picokine ELISA Human-ICAM-1) and Transforming Growth Factor beta (Boster® Picokine ELISA TGF-β) serum levels were also analyzed. The intra- and inter

Article Type:
Research Article
DOI:
https://doi.org/10.1159/000512017
Volume/Issue:
Volume 10: Issue 3
Free access
Caiyan Mo Department of Endocrinology, Beijing Tiantan Hospital, Capital Medical University, Beijing, China

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Han Chen Department of Endocrinology, Beijing Tiantan Hospital, Capital Medical University, Beijing, China

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Qi Zhang Department of Endocrinology, Beijing Tiantan Hospital, Capital Medical University, Beijing, China

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Ying Guo Department of Endocrinology, Beijing Tiantan Hospital, Capital Medical University, Beijing, China

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Liyong Zhong Department of Endocrinology, Beijing Tiantan Hospital, Capital Medical University, Beijing, China

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thyroid cells, and TSH inhibits Fas antigen-mediated apoptosis of thyroid cells and promotes thyroid growth ( 32 ). Intercellular adhesion molecule-1 (ICAM-1) and major histocompatibility complex (MHC) II molecules are thought to be causative factors in

Article Type:
Review Article
Collections:
Hyperthyroidism and thyrotoxicosis
DOI:
https://doi.org/10.1530/ETJ-22-0223
Volume/Issue:
Volume 12: Issue 4
Open access