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Internal Medicine, Erasmus MC, University Medical Center Rotterdam
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Department of Ophthalmology, Geneva University Hospitals, Geneva, Switzerland
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Internal Medicine, Erasmus MC, University Medical Center Rotterdam
Rotterdam Eye Hospital, Rotterdam, The Netherlands
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;47:5197-5203. 10.1167/iovs.06-0596 17122103 19 Valyasevi RW, Jyonouchi SC, Dutton CM, et al: Effect of tumor necrosis factor-alpha, interferon-gamma, and transforming growth factor-beta on adipogenesis and expression of thyrotropin receptor in human orbital
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platelet-derived growth factor receptor beta gene in atypical chronic myeloid leukemia with t(5;10)(q33;q22). Blood 2001;97:3910–3918. 10.1182/blood.V97.12.3910 11389034 76 Puxeddu E, Zhao G, Stringer JR, Medvedovic M, Moretti S, Fagin JA
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, Ohta K , Hamaguchi A , Omura T , Yukimura T , Miura K , et al. Angiotensin II type I receptor antagonist inhibits the gene expression of transforming growth factor-beta 1 and extracellular matrix in cardiac and vascular tissues of hypertensive
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hypertrophy (sonic hedgehog [SHH], GLI; transforming growth factor β [TGFβ], SMAD; hypoxia-inducible factor-1α [HIF1α] and MAPK). Expression of DIO3 is limited by miR-214 and possibly by miR-21 acting through GRHL. See text for details. Tissue TH
Computational Metagenomics, CIBIO Department, University of Trento, Trento, Italy
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-called “dysbiosis” or “dysbacteriosis” could lead to GD and, in extreme cases, GO. Transforming growth factor beta (TGFβ) is secreted by gut dendritic cells and is a differentiation factor for both Th17 and Treg cells, the latter characterised by expression of the
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, Kuijt W, Turano H, Harney J, Larsen PR: Transforming growth factor-β promotes inactivation of extracellular thyroid hormones via transcriptional stimulation of type 3 iodothyronine deiodinase. Mol Endocrinol 2005;19:3126–3136. 10.1210/me.2005
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in sera were analyzed. Serum fibrosis markers such as Intercellular Adhesion Molecule-1 (Boster® Picokine ELISA Human-ICAM-1) and Transforming Growth Factor beta (Boster® Picokine ELISA TGF-β) serum levels were also analyzed. The intra- and inter
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monoclonal in origin [ 15 ], thus suggesting that the development of TSHomas might be explained by the presence of a transforming event providing gain of proliferative function followed by secondary mutations or alterations favoring tumor progression [ 16
Department of Endocrinology and Metabolism, Institute of Endocrinology, NHC Key Laboratory of Diagnosis and Treatment of Thyroid Diseases, The First Affiliated Hospital of China Medical University, Shenyang, China
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-γ, transforming growth factor (TGF)-β) and cortisol levels was performed with ELISA kits (Meimian, Jiangsu, China) as per manual and analyzed under a wavelength of 450 nm. Statistical analysis Data are presented as the median (interquartile range, IQR) or
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Department of Medicine, Monash University, Melbourne, VIC, Australia
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inflammation and fibrosis. Activated T cells produce inflammatory cytokines, including multiple interleukins (IL) such as IL-4, IL-5, IL-10, and IL-13, interferon gamma (INF-γ), and transcription growth factor beta (TGF-β) [ 14 ]. The major function of IL-4 and
