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Carla Colombo Endocrine Oncology Unit, Department of Endocrine and Metabolic Diseases, Istituto Auxologico Italiano IRCCS, Milan, Italy
Department of Pathophysiology and Transplantation, University of Milan, Milan, Italy

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Daniele Ceruti Department of Biotechnology and Translational Medicine, University of Milan, Milan, Italy

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Massimiliano Succi Department of Biotechnology and Translational Medicine, University of Milan, Milan, Italy

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Simone De Leo Endocrine Oncology Unit, Department of Endocrine and Metabolic Diseases, Istituto Auxologico Italiano IRCCS, Milan, Italy

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Matteo Trevisan Endocrine Oncology Unit, Department of Endocrine and Metabolic Diseases, Istituto Auxologico Italiano IRCCS, Milan, Italy
Department of Biotechnology and Translational Medicine, University of Milan, Milan, Italy

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Claudia Moneta Department of Biotechnology and Translational Medicine, University of Milan, Milan, Italy

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Laura Fugazzola Endocrine Oncology Unit, Department of Endocrine and Metabolic Diseases, Istituto Auxologico Italiano IRCCS, Milan, Italy
Department of Pathophysiology and Transplantation, University of Milan, Milan, Italy

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assays have recently become more sensitive and a new cutoff of 15 μg/dL has been proposed ( 23 , 24 , 25 ), we calculated the prevalence of PAI accordingly. To avoid false-positive and/or false-negative results for renin and aldosterone measurement

Article Type:
Research Article
Collections:
Thyroid cancer - basic and translational
DOI:
https://doi.org/10.1530/ETJ-23-0246
Volume/Issue:
Volume 13: Issue 3
Open access
Toshihide Yamamoto Yao Tokushukai General Hospital, Yao, Japan

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LDT at 30 min was judged by a CL set at 497 nmol/l [ 16 ]. The assays of levels of cortisol, ACTH, aldosterone, TSH, and free thyroxine were performed by a commercial laboratory (Special Reference Laboratories SRL, Hachioji, Japan). Blood for

Article Type:
Case Report
DOI:
https://doi.org/10.1159/000433532
Volume/Issue:
Volume 4: Issue 3
Free access
Luba Freja Michaelsson Department of Endocrinology, Herlev Hospital, University of Copenhagen, Copenhagen, Denmark

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Jeppe Lerche  la Cour Department of Endocrinology, Herlev Hospital, University of Copenhagen, Copenhagen, Denmark

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Bjarke Borregaard Medici Department of Endocrinology, Herlev Hospital, University of Copenhagen, Copenhagen, Denmark

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Torquil Watt Department of Endocrinology, Herlev Hospital, University of Copenhagen, Copenhagen, Denmark

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Jens Faber Department of Endocrinology, Herlev Hospital, University of Copenhagen, Copenhagen, Denmark
Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark

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Birte Nygaard Department of Endocrinology, Herlev Hospital, University of Copenhagen, Copenhagen, Denmark
Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark

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-angiotensin system activity. J Renin Angiotensin Aldosterone Syst 2009; 10: 216–229. 14 Cramon P, Bonnema SJ, Bjorner JB, Ekholm O, Feldt-Rasmussen U, Frendl DM, Groenvold M, Hegedus L, Rasmussen AK, Watt T: Quality of life in patients with benign nontoxic

Article Type:
Research Article
DOI:
https://doi.org/10.1159/000490383
Volume/Issue:
Volume 7: Issue 5
Free access
João Sérgio Neves Unidade de Investigação Cardiovascular, Departamento de Cirurgia e Fisiologia, Faculdade de Medicina da Universidade do Porto, Porto, Portugal
Department of Endocrinology, Diabetes and Metabolism, Centro Hospitalar Universitário de São João, Porto, Portugal

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Ricardo Fontes-Carvalho Unidade de Investigação Cardiovascular, Departamento de Cirurgia e Fisiologia, Faculdade de Medicina da Universidade do Porto, Porto, Portugal
Department of Cardiology, Centro Hospitalar Gaia/Espinho, Vila Nova de Gaia, Portugal

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Marta Borges-Canha Unidade de Investigação Cardiovascular, Departamento de Cirurgia e Fisiologia, Faculdade de Medicina da Universidade do Porto, Porto, Portugal
Department of Endocrinology, Diabetes and Metabolism, Centro Hospitalar Universitário de São João, Porto, Portugal

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Ana Rita Leite Unidade de Investigação Cardiovascular, Departamento de Cirurgia e Fisiologia, Faculdade de Medicina da Universidade do Porto, Porto, Portugal

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Sandra Martins Department of Clinical Pathology, Centro Hospitalar Universitário de São João, Porto, Portugal
EPIUnit – Instituto de Saúde Pública, Universidade do Porto, Porto, Portugal

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Ana Oliveira Department of Endocrinology, Diabetes and Metabolism, Centro Hospitalar Universitário de São João, Porto, Portugal

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João Tiago Guimarães Department of Clinical Pathology, Centro Hospitalar Universitário de São João, Porto, Portugal
Department of Biomedicine, Faculdade de Medicina da Universidade do Porto, Porto, Portugal
EPIUnit – Instituto de Saúde Pública, Universidade do Porto, Porto, Portugal

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Davide Carvalho Department of Endocrinology, Diabetes and Metabolism, Centro Hospitalar Universitário de São João, Porto, Portugal
Instituto de Investigação e Inovação em Saúde (i3S), Universidade do Porto, Porto, Portugal

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Adelino Leite-Moreira Unidade de Investigação Cardiovascular, Departamento de Cirurgia e Fisiologia, Faculdade de Medicina da Universidade do Porto, Porto, Portugal

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Ana Azevedo EPIUnit – Instituto de Saúde Pública, Universidade do Porto, Porto, Portugal
Departamento de Ciências da Saúde Pública e Forenses e Educação Médica, Faculdade de Medicina da Universidade do Porto, Porto, Portugal

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tachycardic effects appear to be dependent on direct cardiac electrophysiological effects [ 49 - 51 ] and β-adrenergic stimulation [ 52 ], while the volume overload is probably mediated by peripheral vasodilation with compensatory activation of the renin-angiotensin-aldosterone

Article Type:
Research Article
DOI:
https://doi.org/10.1159/000508407
Volume/Issue:
Volume 10: Issue 2
Free access
E.N. Klein Hesselink Department of Endocrinology, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands

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T.P. Links Department of Endocrinology, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands

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, high triiodothyronine levels enhance cardiac contractility. Furthermore, triiodothyronine lowers systemic vascular resistance by acting on the vascular smooth muscle cells. Via the renin-angiotensin-aldosterone system, blood volume and therefore cardiac

Article Type:
Review Article
DOI:
https://doi.org/10.1159/000432397
Volume/Issue:
Volume 4: Issue 2
Free access