Department of Endocrinology and Metabolism, Institute of Endocrinology, NHC Key Laboratory of Diagnosis and Treatment of Thyroid Diseases, The First Affiliated Hospital of China Medical University, Shenyang, China
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Introduction Autoimmune thyroiditis (AIT), also referred to as Hashimoto’s thyroiditis (HT) or chronic lymphocytic thyroiditis, is a common autoimmune disorder with a prevalence of 0.3–1.5 cases per 1000 subjects per year ( 1 ). AIT is
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, Hashimoto thyroiditis has become the archetype of autoimmune destructive disorders and autoantibody production. This lecture honours Professor Aldo Pinchera, who has made many important contributions to our understanding of thyroid autoimmunity; remarkably
INSERM 1086 ANTICIPE, Caen University, Caen, France
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prostate cancer and were related to chronic autoimmune thyroiditis (CAT) ( 4 , 5 ). Diffuse thyroid uptake is also a well-known pattern in patients with CAT on 18 F-Fluorodeoxyglucose ( 18 FDG) PET/CT ( 6 , 7 ). Although some authors showed that thyroid
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factors (i.e., sex, age, and cigarette smoking) [ 11 - 14 ], pathological conditions (i.e., small-cell lung carcinoma, chronic renal failure, hypergastrinemia, neuroendocrine tumors, autoimmune thyroiditis (AIT), and micropapillary thyroid carcinoma) [ 15
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Introduction Autoimmune thyroiditis (AIT) is a chronic inflammation of the thyroid gland, which affects 10% of females and 2% of the male population [ 1 ]. An inverse correlation between serum selenium concentration and the incidence of AIT
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for a familial history of autoimmune thyroiditis. He complained of mild asthenia. His brother was affected by OCA and autoimmune thyroiditis. The physical examination revealed the presence of fair skin, platinum white hair, hypopigmentation of the iris
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corticosteroids in the short term, and the term ‘steroid-responsive encephalopathy associated with autoimmune thyroiditis' is also used to describe this disease [ 1 ]. We report on a patient with HE with a significant clinical improvement correlating with a 20
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recognize only noniodinated Tg epitopes [ 21 ]. Furthermore, in experimental models of autoimmune thyroiditis, it has been shown that breaking B-cell self-tolerance occurs first for Tg and subsequently for thyroperoxidase [ 26 ]. It is not known how Tg
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to differentiate between thyrotoxicosis due to destructive thyroiditis and autoimmune hyperthyroidism ensuing during ICPi treatment are: severity (mild to moderate vs. moderate to severe), duration (self-limited vs. persistent), lag time (weeks vs
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Images in thyroidology Clinical and laboratory findings of subacute thyroiditis have been repeatedly reported as being associated with acute Sars-Cov-2 infection and post-COVID-19 syndrome ( 1 , 2 , 3 ). Exact mechanisms and