Salman Razvi
Salman Razvi
Translational and Clinical Research Institute, Newcastle University, Newcastle upon Tyne, UK
Department of Endocrinology, Gateshead Health NHS Foundation Trust, Gateshead, UK
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Department of Endocrinology, Gateshead Health NHS Foundation Trust, Gateshead, UK
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Avais Jabbar
Avais Jabbar
Translational and Clinical Research Institute, Newcastle University, Newcastle upon Tyne, UK
Department of Cardiology, Newcastle upon Tyne Hospitals NHS Foundation Trust, Newcastle upon Tyne, UK
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Department of Cardiology, Newcastle upon Tyne Hospitals NHS Foundation Trust, Newcastle upon Tyne, UK
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Arjola Bano
Arjola Bano
Department of Cardiology, Institute of Social and Preventive Medicine, University of Bern, Bern University Hospital, Bern, Switzerland
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Lorna Ingoe
Lorna Ingoe
Department of Endocrinology, Gateshead Health NHS Foundation Trust, Gateshead, UK
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Peter Carey
Peter Carey
Departments of Endocrinology and Cardiology, South Tyneside and Sunderland NHS Foundation Trust, Sunderland, UK
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Shahid Junejo
Shahid Junejo
Departments of Endocrinology and Cardiology, South Tyneside and Sunderland NHS Foundation Trust, Sunderland, UK
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Honey Thomas
Honey Thomas
Department of Cardiology, Northumbria Healthcare NHS Foundation Trust, Cramlington, UK
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Caroline Addison
Caroline Addison
Department of Biochemistry, Gateshead Health NHS Foundation Trust, Gateshead, UK
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David Austin
David Austin
Department of Cardiology, South Tees Health NHS Foundation Trust, Middlesbrough, UK
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John P Greenwood
John P Greenwood
Leeds Institute of Cardiovascular and Metabolic Medicine, University of Leeds, Leeds, UK
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Azfar G Zaman
Azfar G Zaman
Translational and Clinical Research Institute, Newcastle University, Newcastle upon Tyne, UK
Department of Cardiology, Newcastle upon Tyne Hospitals NHS Foundation Trust, Newcastle upon Tyne, UK
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Department of Cardiology, Newcastle upon Tyne Hospitals NHS Foundation Trust, Newcastle upon Tyne, UK
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high ( 1 ). Therefore, additional strategies are required to further improve the prognosis and outcomes of AMI patients. Clinical and experimental data support an additional critical role for inflammation in the pathophysiology of plaque instability and
Victor J M Pop
Victor J M Pop
Department of Medical Psychology, Tilburg University, The Netherlands
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Johannes G Krabbe
Johannes G Krabbe
Department of Clinical Chemistry and Laboratory Medicine, Medisch Spectrum Twente, Medlon BV, Enschede, The Netherlands
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Maarten Broeren
Maarten Broeren
Department of Clinical Chemistry, Maxima Medical Centre, Veldhoven, The Netherlands
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Wilmar Wiersinga
Wilmar Wiersinga
Department of Endocrinology and Metabolism, Academic Medical Center, University of Amsterdam, The Netherlands
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Margaret P Rayman
Margaret P Rayman
Department of Nutritional Sciences, Faculty of Health and Medical Sciences, University of Surrey, Guildford, UK
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. Conception and implantation are characterized by a state of physiological inflammation accompanied by a heightened response of T-helper cells (Th1 and Th2) resulting in the production of inflammatory cytokines including IL-6 and IL-8 ( 10 ). hCG is important
Jinrong Fu
Jinrong Fu
Department of Endocrinology, Guangdong Provincial People's Hospital (Guangdong Academy of Medical Sciences), Southern Medical University, Guangzhou, China
Department of Endocrinology and Metabolism, Institute of Endocrinology, NHC Key Laboratory of Diagnosis and Treatment of Thyroid Diseases, The First Affiliated Hospital of China Medical University, Shenyang, China
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Department of Endocrinology and Metabolism, Institute of Endocrinology, NHC Key Laboratory of Diagnosis and Treatment of Thyroid Diseases, The First Affiliated Hospital of China Medical University, Shenyang, China
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Zihao Fan
Zihao Fan
Department of Geriatrics, Guangdong Provincial Geriatrics Institute, Guangdong Provincial People's Hospital (Guangdong Academy of Medical Sciences), Southern Medical University, Guangzhou, China
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Liang He
Liang He
Department of Thyroid Surgery, The First Affiliated Hospital of China Medical University, Shenyang, China
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Qian Liu
Qian Liu
Department of Endocrinology and Metabolism, Jilin Cancer Hospital, Changchun, Jilin, China
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He Liu
He Liu
Department of Endocrinology and Metabolism, Institute of Endocrinology, NHC Key Laboratory of Diagnosis and Treatment of Thyroid Diseases, The First Affiliated Hospital of China Medical University, Shenyang, China
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Yushu Li
Yushu Li
Department of Endocrinology and Metabolism, Institute of Endocrinology, NHC Key Laboratory of Diagnosis and Treatment of Thyroid Diseases, The First Affiliated Hospital of China Medical University, Shenyang, China
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Haixia Guan
Haixia Guan
Department of Endocrinology, Guangdong Provincial People's Hospital (Guangdong Academy of Medical Sciences), Southern Medical University, Guangzhou, China
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inflammation in the development of AIT. To test this hypothesis, we examined the circadian characteristics in AIT and investigated the effects of light shift (LS) on thyroid inflammation. Materials and methods Recruitment of patients and sample
Eveline Bruinstroop
Eveline Bruinstroop
Department of Endocrinology and Metabolism, Amsterdam UMC, University of Amsterdam, Amsterdam, the Netherlands
Amsterdam Gastroenterology Endocrinology Metabolism, Amsterdam, the Netherlands
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Amsterdam Gastroenterology Endocrinology Metabolism, Amsterdam, the Netherlands
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Anne H van der Spek
Anne H van der Spek
Department of Endocrinology and Metabolism, Amsterdam UMC, University of Amsterdam, Amsterdam, the Netherlands
Amsterdam Gastroenterology Endocrinology Metabolism, Amsterdam, the Netherlands
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Amsterdam Gastroenterology Endocrinology Metabolism, Amsterdam, the Netherlands
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Anita Boelen
Anita Boelen
Amsterdam Gastroenterology Endocrinology Metabolism, Amsterdam, the Netherlands
Department of Laboratory Medicine, Endocrine Laboratory, Amsterdam UMC, University of Amsterdam, Amsterdam, the Netherlands
Amsterdam Reproduction & Development Research Institute, Amsterdam, the Netherlands
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Department of Laboratory Medicine, Endocrine Laboratory, Amsterdam UMC, University of Amsterdam, Amsterdam, the Netherlands
Amsterdam Reproduction & Development Research Institute, Amsterdam, the Netherlands
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liver are T3-related. Role of liver deiodinases in liver disease The function of the normal liver can be altered by diseases involving liver inflammation (hepatitis), scarring (fibrosis), and cancer (hepatocellular carcinoma). Different cell
Carla Gambale
Carla Gambale
Department of Clinical and Experimental Medicine, Unit of Endocrinology, Pisa University Hospital, Pisa, Italy
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Alessandro Prete
Alessandro Prete
Department of Clinical and Experimental Medicine, Unit of Endocrinology, Pisa University Hospital, Pisa, Italy
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Chiara Romei
Chiara Romei
Department of Diagnostic Imaging, Unit of Radiology, Pisa University Hospital, Pisa, Italy
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Alessandro Celi
Alessandro Celi
Department of Surgery, Medicine, Molecular Biology and Critical Care, Respiratory Pathophysiology Unit, Pisa University Hospital, Pisa, Italy
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Rossella Elisei
Rossella Elisei
Department of Clinical and Experimental Medicine, Unit of Endocrinology, Pisa University Hospital, Pisa, Italy
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Antonio Matrone
Antonio Matrone
Department of Clinical and Experimental Medicine, Unit of Endocrinology, Pisa University Hospital, Pisa, Italy
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, pulmonary toxicity potentially linked to selpercatinib treatment could be more heterogeneous than conceived in the past, including also pulmonary AEs often observed with other MKIs. OB is characterized by inflammation and fibrosis in conducting airways
Francesco Boi
Francesco Boi
Endocrinology Unit, Department of Medical Sciences and Public Health, University of Cagliari, Cagliari, Italy
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Fabiana Pani
Fabiana Pani
Endocrinology Unit, Department of Medical Sciences and Public Health, University of Cagliari, Cagliari, Italy
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Stefano Mariotti
Stefano Mariotti
Endocrinology Unit, Department of Medical Sciences and Public Health, University of Cagliari, Cagliari, Italy
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summarize the clinical and biological evidence supporting this link. Background: Chronic Inflammation and Thyroid Cancer Inflammatory or infectious diseases may be strongly associated with human tumors. Several inflammatory/immune cells, such as
G. Teckie
G. Teckie
Divisions of Endocrinology, Chris Hani Baragwanath Academic Hospital and University of the Witwatersrand, Johannesburg, South Africa
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S.A. Bhana
S.A. Bhana
Divisions of Endocrinology, Chris Hani Baragwanath Academic Hospital and University of the Witwatersrand, Johannesburg, South Africa
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J.M.L. Tsitsi
J.M.L. Tsitsi
Infectious Diseases, Chris Hani Baragwanath Academic Hospital and University of the Witwatersrand, Johannesburg, South Africa
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R. Shires
R. Shires
Divisions of Endocrinology, Chris Hani Baragwanath Academic Hospital and University of the Witwatersrand, Johannesburg, South Africa
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thyroid hormone is released into the circulation as a result of inflammation and disruption of the thyroid follicles [ 14 ]. Even when only part of the gland is affected, radioactive iodine uptake may be suppressed due, in part at least, to the inhibition
Pauline Campredon
Pauline Campredon
Department of Endocrinology and Metabolic Diseases, Larrey University Hospital, Toulouse, France
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Philippe Imbert
Philippe Imbert
Ophthalmologist, Clinique du Parc, Toulouse, France
Multidisciplinary Thyroid Eye Consultation, Department of Endocrinology, Larrey University Hospital, Toulouse, France
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Multidisciplinary Thyroid Eye Consultation, Department of Endocrinology, Larrey University Hospital, Toulouse, France
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Céline Mouly
Céline Mouly
Department of Endocrinology and Metabolic Diseases, Larrey University Hospital, Toulouse, France
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Solange Grunenwald
Solange Grunenwald
Department of Endocrinology and Metabolic Diseases, Larrey University Hospital, Toulouse, France
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Julien Mazières
Julien Mazières
Department of Pneumology, Larrey University Hospital, Toulouse, France
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Philippe Caron
Philippe Caron
Department of Endocrinology and Metabolic Diseases, Larrey University Hospital, Toulouse, France
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confirmed orbital fat expansion (Fig. 2 b) and showed inflammation on T2-weighted images similar to thyroid-like ophthalmopathy. Clinical workup found no evidence for myasthenia gravis (absence of anti-acetylcholine receptor antibody), orbital inflammatory
Sita Virakul
Sita Virakul
Departments of Immunology, University Medical Center Rotterdam
Internal Medicine, Erasmus MC, University Medical Center Rotterdam
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Internal Medicine, Erasmus MC, University Medical Center Rotterdam
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Leendert van Steensel
Leendert van Steensel
Departments of Immunology, University Medical Center Rotterdam
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Virgil A.S.H. Dalm
Virgil A.S.H. Dalm
Departments of Immunology, University Medical Center Rotterdam
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Dion Paridaens
Dion Paridaens
Rotterdam Eye Hospital, Rotterdam, The Netherlands
Department of Ophthalmology, Geneva University Hospitals, Geneva, Switzerland
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Department of Ophthalmology, Geneva University Hospitals, Geneva, Switzerland
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P. Martin van Hagen
P. Martin van Hagen
Departments of Immunology, University Medical Center Rotterdam
Internal Medicine, Erasmus MC, University Medical Center Rotterdam
Rotterdam Eye Hospital, Rotterdam, The Netherlands
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Internal Medicine, Erasmus MC, University Medical Center Rotterdam
Rotterdam Eye Hospital, Rotterdam, The Netherlands
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Willem A. Dik
Willem A. Dik
Departments of Immunology, University Medical Center Rotterdam
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therapeutic options to target the PDGF-system in GO. The Pathophysiology of GO The pathophysiology of GO comprises various cell types, inflammatory mediators and autoantibodies that interact with each other, resulting in orbital inflammation and
Georgios K. Markantes
Georgios K. Markantes
Division of Endocrinology – Department of Internal Medicine, University of Patras Medical School, Patras, Greece
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Marina A. Michalaki
Marina A. Michalaki
Division of Endocrinology – Department of Internal Medicine, University of Patras Medical School, Patras, Greece
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George A. Vagenakis
George A. Vagenakis
Department of Pediatric Cardiology and Adult Congenital Heart Disease, Onassis Cardiac Surgery Center, Athens, Greece
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Fotini N. Lamari
Fotini N. Lamari
Laboratory of Pharmacognosy and Chemistry of Natural Products, Department of Pharmacy, University of Patras, Patras, Greece
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Efthymia Pitsi
Efthymia Pitsi
Laboratory of Pharmacognosy and Chemistry of Natural Products, Department of Pharmacy, University of Patras, Patras, Greece
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Maria Eliopoulou
Maria Eliopoulou
Endocrinology Unit, Karamandanio Hospital, Patras, Greece
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Nicholas G. Beratis
Nicholas G. Beratis
Department of Pediatrics, University of Patras Medical School, Patras, Greece
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Kostas B. Markou
Kostas B. Markou
Division of Endocrinology – Department of Internal Medicine, University of Patras Medical School, Patras, Greece
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in AIT2 [ 6 ]. Serum levels of interleukin-6 (IL-6) have been used as a marker of thyroid inflammation and high levels may support the diagnosis of AIT2, but poor specificity and high cost limit its routine use [ 7 , 8 ]. Beta-glucuronidase (β
